LEI 11418 PDF

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In the latter case, please turn on Javascript support in your web browser and reload this page. Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease.

Or filter your current search. Or filter your current search. CitePeer Related Articles http: In the latter case, please turn on Javascript support in your web browser and reload this page. Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually be mediated by modulation of MAPK signaling cascades.

Read Article at publisher’s site. Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription.

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Gene Ontology GO Terms. Abstract Our aim is to investigate the potential therapeutic value of morin against osteoporosis and elucidate the mechanism of action. How does Europe PMC derive its citations network?

Journal of Natural Medicines [03 Sep65 1: We investigated the effects of GBE on glucose consumption, glucokinase activity, and mRNA levels of key genes in glucose metabolism and the insulin signaling pathway.

Our data suggest that GBE has the potential to prevent insulin resistance and is a promising anti-diabetic drug.

Find all citations in this journal default. Body mineral density BMD was determined at proximal femurs using dual energy X-ray absorptiometry.

Either your web browser doesn’t support Javascript or it is currently turned off. Europe PMC requires Javascript to function effectively.

It is said that GBE treatment could avoid drug-induced obesity. Find all citations in this journal default. Body weight was regularly monitored. Morin treatment also increased the number of trabecular bones in DEX-induced rats. Abstract Ginkgo biloba, an herbal medication, is capable of lowering glucose, fat, and lipid peroxide in diabetic patients.

Read Article at publisher’s site.

To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of thiazolidinedione TZD. Journal of Natural Medicines [24 Jul72 4: The data indicated that GBE repressed glucose uptake under normal conditions, while it dramatically improved glucose tolerance under insulin-resistant conditions.

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Pathological examination was performed by hematoxylin and eosin staining. It should be noted 11481, unlike rosiglitazone, GBE did not stimulate excessive glucose uptake 11148 it improved glucose tolerance.

Body weight and BMD were both significantly decreased in osteoporotic rats, although BMD was partially restored by intraperitoneal morin administration.

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Gene Ontology GO Terms. In the current study, we leu the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes. The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively. How does Europe PMC derive its citations network? Either your web browser doesn’t support Javascript or it is currently turned off.

CitePeer Related Articles http: Europe PMC requires Javascript to function effectively.